参考文献[1] 林鹏, 郭新星, 魏竹亮. 局部注射黄芪多糖对大鼠正畸牙保持阶段BMP-2表达的影响[J]. 口腔医学研究, 2017, 33(5):520-524.[2] 武密山,赵素芝,任立中等. 补骨脂对成骨细胞核因子-κβ 受体激活配体和骨保护素表达的影响[J]. 中国老年学杂志,2012,32(1):66-69.[3] 李启活. 补骨脂素激活MAPK-NF-κB信号通路促进成骨细胞增殖的研究[D]. 广州中医药大学,2016.[4] 王建华,王艳,潘永梅. 补骨脂素对大鼠成骨细胞增殖和分化的影响[J]. 天然产物研究与开发,2007,19(5):844-846.[5] Kang W-b, Deng Y-t, Wang D-s, et al. Osteoprotective effects of estrogen membrane receptor GPR30 in ovariectomized rats[J]. Journal of Steroid Biochemistry and Molecular Biology, 2015, 154: 237-244.[6] Luo LJ, Liu F, Lin ZK, et al. Genistein regulates the IL-1 beta induced activation of MAPKs in human periodontal ligament cells through G protein-coupled receptor 30[J]. Arch Biochem Biophys, 2012, 522(1): 9-16.[7] 韩光红,陈远萍,刘天悦等.正畸牙齿复发后牙周组织改建的实验研究[J].口腔医学研究,2010,(04):508-511.[8] Ashizawa Y, Sahara N. Quantitative evaluation of newly formed bone in the alveolar wall surrounding the root during the initial stage of experimental tooth movement in the rat[J]. Archives of Oral Biology, 1998, 43(6): 473-484.[9] 章魁华,于世凤. 实验口腔医学[M].北京:人民卫生出版,2009;3-14[10] 刘世颖,陈远萍,刘天悦等. 实验性正畸牙移动保持期动物模型的建立[J].中国实用口腔科杂志,2009,(09):537-539.[11] King GJ, Keeling SD, McCoy EA, et al. MEASURING DENTAL DRIFT AND ORTHODONTIC TOOTH MOVEMENT IN RESPONSE TO VARIOUS INITIAL FORCES IN ADULT-RATS[J]. American Journal of Orthodontics and Dentofacial Orthopedics, 1991, 99(5): 456-465.[12] 杨琳,曾英,李劲平等. 补骨脂素对去势雌鼠E_2、ER_β、TNF-α、IL-17的影响[J]. 中国骨质疏松杂志,2016,(04):387-392+395.[13] Yuan X, Bi Y, Yan Z, et al. Psoralen and Isopsoralen Ameliorate Sex Hormone Deficiency-Induced Osteoporosis in Female and Male Mice[J]. BioMed research international, 2016, 2016: 6869452.[14] 杨召,杨华,姚振强等. 补肾中药对去卵巢大鼠骨组织ER、OPG表达的促进作用[J]. 中医正骨,2004,(03):3-5+63.[15] Yang Z, Huang JH, Liu SF, et al. The osteoprotective effect of psoralen ovariectomy-induced osteoporotic rats via stimulating the osteoblastic differentiation from bone mesenchymal stem cells[J]. Menopause, 2012, 19(10): 1156-1164.[16] Xu K, Pan X, Sun YJ, et al. Psoralen activates cartilaginous cellular functions of rat chondrocytes in vitro[J]. Pharmaceutical Biology, 2015, 53(7): 1010-1015.[17] 丁一,杨恒,文钦. 补肾固齿丸对大鼠实验性牙周炎牙槽骨重建的影响[J]. 第三军医大学学报,2011,(22):2390-2393.[18] Han G, Chen Y, Hou J, et al. Effects of simvastatin on relapse and remodeling of periodontal tissues after tooth movement in rats[J]. Am J Orthod Dentofacial Orthop, 2010, 138(5): 550 e551-557; discussion 550-551.[19] Wang Y, Wang XX, Zhang LN, et al. Effects of traditional Chinese medicine on bone remodeling during orthodontic tooth movement[J]. J Ethnopharmacol, 2012, 141(2): 642-646.[20] Fujimura Y, Kitaura H, Yoshimatsu M, et al. Influence of bisphosphonates on orthodontic tooth movement in mice.[J]. European Journal of Orthodontics, 2009, 31(6):572.[21] Hirate Y, Yamaguchi M, Kasai K. Effects of Relaxin on Relapse and Periodontal Tissue Remodeling after Experimental Tooth Movement in Rats[J]. Connective Tissue Research, 2011, 53(3): 207-219.[22] Zhao M, Xiao GZ, Berry JE, et al. Bone morphogenetic protein 2 induces dental follicle cells to differentiate toward a cementoblast/osteoblast phenotype[J]. Journal of Bone and Mineral Research, 2002, 17(8): 1441-1451.[23] Kemoun P, Laurencin-Dalicieux S, Rue J, et al. Human dental follicle cells acquire cementoblast features under stimulation by BMP-2/-7 and enamel matrix derivatives (EMD) in vitro[J]. Cell and Tissue Research, 2007, 329(2): 283-294.[24] Zeng J, Wang X-x, Jia Y-l, et al. Effect of pamidronate on cementoclast and osteoclast differentiation factor expression during orthodontic tooth movement in rats[J]. Shanghai kou qiang yi xue = Shanghai journal of stomatology, 2012, 21(5): 495-500.[25] Windahl SH, Andersson N, Martensson UEA, et al. Deficiency of the G protein-coupled estrogen receptor GPR30 leads to reduced bone growth and bone mineral density in female mice[J]. Journal of Bone and Mineral Research, 2007, 22: S280-S280. |