[1]王琳源,林晓萍,靳赢.适应性免疫应答在牙周炎中的作用[J].中华口腔医学, 2013, 48(2):115-118
[2]王琳源,林晓萍,靳赢.细胞在不同菌株诱导小鼠牙周炎中的表达[J].上海口腔医学, 2014, 23(1):20-25
[3]Cheng WC1, Hughes FJ, Taams LS.The presence,function and regulation of IL-17 and Th17 cells in periodontitis[J].J Clin Periodontol, 2014, 41(6):541-549
[4]Lee W, Lee GR.Transcriptional regulation and development of regulatory T cells[J].Exp Mol Med, 2018, 50(3):e456-e456
[5]Harrington LE, Hatton RD, Mangan PR, et al.Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages[J].Nat Immunol, 2005, 6(11):1123-1132
[6]Park H, Li Z, Yang XO, et al.A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17[J].Nat Immunol, 2005, 6(11):1133-1141
[7]Wacleche VS, Landay A, ?Routy JP, et al.The Th17 Lineage: From Barrier Surfaces Homeostasis to Autoimmunity,Cancer,and HIV-1 Pathogenesis[J].Viruses, 2017, 9(10):e303-e303
[8]Harrington LE, Hatton RD, Mangan PR.Interlenkin 17- producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages[J].Nat Immunol, 2005, 6(11):1123-1132
[9]Korn T, Bettelli E, Gao W, et al.IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells[J].Nature, 2007, 448(7152):484-487
[10]Bettelli E, Carrier Y, Gao W, et al.Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells[J].Nature, 2006, 441(7090):235-238
[11]Imamura E, Taguchi K, Sasaki-Iwaoka H, et al.Anti-IL-23?receptor monoclonal antibody prevents CD4+ T cell-mediated colitis in association with decreased systemic Th1 and?Th17?responses[J].Eur J Pharmacol, 2018, 824:163-169
[12]McGeachy MJ, ?Bak-Jensen KS, Chen Y, et al.TGF-beta and IL-6 drive the production of IL-17 and IL-10 by T cells and restrain T(H)-17 cell-mediated pathology[J].Nat Immunol, 2007, 8(12):1390-1397
[13]王琳源,林晓萍,靳赢.适应性免疫应答在牙周炎中的作用[J].中华口腔医学, 2013, 48(2):115-118
[14]Bittner-Eddy PD, Fischer LA, Kaplan DH, et al.Mucosal Langerhans Cells Promote Differentiation of Th17 Cells in a Murine Model of Periodontitis but Are Not Required for Porphyromonas gingivalis-Driven Alveolar BoneDestruction[J].J Immunol, 2016, 197(4):1435-1446
[15]Cheng WC, van Asten SD, Burns LA, et al.Periodontitis-associated pathogens Pgingivalis and A. actinomycetemcomitans activate human CD14(+) monocytes leading to enhanced Th17IL-17?responses[J].Eur J Immunol, 2016, 46(9):2211-2221
[16]Song X, Qian Y.The activation and regulation of IL-17 receptor mediated signaling[J].Cytokine, 2013, 62(2):175-182
[17]Takahashi N, Okui T, Tabeta K, et al.Effect of interleukin-17 on the expression of chemokines in gingival epithelial cells[J].Eur J Oral Sci, 2011, 119(5):339-344
[18]王琳源,关宁,林晓萍.辅助性细胞在牙周炎小鼠中的免疫状态研究[J].华西口腔医学, 2015, 33(2):125-129
[19]Mitani A1, Niedbala W, Fujimura T, et al.Increased expression of interleukin (IL)-35 and IL-17,but not IL-27,in gingival tissues with chronic periodontitis[J].J Periodontol, 2015, 86(2):301-309
[20]Chen XT, Chen LL, Tan JY, et al.Th17 and Th1 Lymphocytes Are Correlated with Chronic Periodontitis[J].Immunol Invest, 2016, 45(3):243-254
[21]Zhao L, Zhou Y, Xu Y, et al.Effect of non-surgical periodontal therapy on the levels of Th17Th1Th2 cytokines and their transcription factors in Chinese chronic periodontitis patients[J].J clin Periodontal, 2011, 38(6):509-516
[22]王琳源,林晓萍.在牙周炎骨吸收中的研究进展[J].口腔医学, 2013, 33(7):493-495
[23]Dutzan N, Gamonal J, Silva A, et al.Over-expression of forkhead box P3 and its association with receptor activator of nuclear factor-kappa B ligand,interleukin (IL) -17,IL-10 and transforming growth factor-beta during the progression of chronic periodontitis[J].J Clin Periodontol, 2009, 36(5):396-403
[24]Kuritani M, Sakai N, Karakawa A, et al.Anti-mouse RANKL Antibodies Inhibit Alveolar Bone Destruction in Periodontitis Model Mice[J].Biol Pharm Bull, 2018, 41(4):637-643
[25]Peters A, Lee Y, Kuchroo VK.The many faces of Th17 cells[J].Curr Opin Immunol, 2011, 23(6):702-706
[26]Pacifici R.The Role of?IL-17?and?TH17 Cells?in the Bone Catabolic Activity of PTH [J].Front Immunol, 2016, 7:57-57
[27]Pollinger B, Junt T, Metzler B, et al.Th17 cells,not IL-17+ γδ T cells,drive arthritic bone destruction in mice and humans[J].J Immunol, 2011, 186(4):2602-2612
[28]Cardoso CR, Garlet GP, Crippa GE, et al.Evidence of the presence of T helper type 17 cells in chronic lesions of human periodontal disease[J].Oral Microbiol Immunol, 2009, 24(1):1-6
[29]Cardoso CR,?Garlet GP,?Crippa GE, et al.Evidence of the presence of T helper type 17 cells in chronic lesions of human periodontal disease[J].Oral Microbiol Immunol, 2009, 24(1):1-6
[30]王琳源,关宁,林晓萍.细胞介导的免疫应答在牙周炎小鼠中的研究[J].实用口腔医学, 2015, 31(3):318-322
[31]Araujo-Pires AC, Vieira AE, Francisconi CF, et al.IL-4CCL22CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis[J].J Bone Miner Res, 2015, 30(3):412-422
[32]Keswani T,?Sarkar S,?Sengupta A, et al.Role of?TGF-β and?IL-6?in dendritic?cells,?Treg?and?Th17?mediated immune response during experimental cerebral malaria[J]. Cytokine, 2016, 88:154-166
[33]Cortes-Perez NG, Lozano-Ojalvo D, Maiga MA, et al.Intragastric administration of Lactobacillus casei BL23 induces regulatory FoxP3+RORγt+ T cells subset in mice[J].Benef Microbes, 2017, 8(3):433-438
[34]Ren J,?Li B.The Functional Stability of?FOXP3?and RORγt in?Treg?and Th17 and Their Therapeutic Applications[J].Adv Protein Chem Struct Biol, 2017, 107:155-189
[35]Kluger MA, Nosko A, Ramcke T, et al.RORγt expression in Tregs promotes systemic lupus erythematosus via IL-17 secretion,alteration of Treg phenotype and suppression of Th2 responses[J].Clin Exp Immunol, 2017, 188(1):63-78
[36]Weitzmann MN.Bone and the Immune System[J].Toxicol Pathol, 2017, 45(7):911-924
[37]Ozaki Y, Koide M, Furuya Y, et al.Treatment of OPG-deficient mice with WP9QY,a RANKL-binding peptide,recovers alveolar bone loss by suppressing osteoclastogenesis and enhancing osteoblastogenesis[J].PLoS One, 2017, 12(9):e0184904-e0184904
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