| [1]张志愿.口腔颌面外科学[M]. 7版.北京：人民卫生出版社,2012：375-376[2]余元勋.中国疾病信号通路与靶向治疗学[M].合肥：安徽科学技术出版社,2013:01-02[3]Klose R,Berger C,Moll I,et al.Soluble Notch ligand and receptor peptides act antagonistically during angiogenesis[J].Cardiovasc Res,2015,107(1):153-163[4]Shimojo H,Ohtsuka T,Kageyama R.Oscillations in notch signaling regulate maintenance of neural progenitors[J].Neuron,2008,58(1):52-64[5]Karlsson C,Jonsson M,Asp J,et al.Notch and HES5 are regulated during human cartilage differentiation[J].Cell T3 Res,2007,327(3):539-551[6]赵兵杰.HIF-1/Notch信号通路在颞下颌关节骨关节炎的研究[D]. 山东大学口腔临床医学,2015[7]Stone AV,Loeser RF,Vanderman KS,et al.Pro-inflammatory stimulation of meniscus cells increases production of matrix metalloproteinases and additional catabolic factors involved in osteoarthritis pathogenesis[J].OsteoarthrCartil,2014,22(2):264-274[8]Wu G,Chen L,Wei G,et al.Effects of sleep deprivation on pain-related factors in the temporomandibular joint[J].J Surg Res,2014,192(1):103-111[9]Palm E,Demirel I,Bengtsson T R,et al.The role of toll-like and protease-activated receptors in the expression of cytokines by gingival fibroblasts stimulated with the periodontal pathogen Porphyromonas gingivalis[J].Cytokine,2015,76(2):424-432[10]Medvedev AE.Toll-like receptor polymorphisms,inflammatory and infectious diseases,allergies,and cancer[J].J Interferon Cytokine Res,2013,33(9):467-484[11]Johnston DG,Corr SC.Toll-Like Receptor Signalling and the Control of Intestinal Barrier Function[J].Methods MolBiol,2016,1390:287-300[12]Ribeiro-Dasilva MC,Fillingim RB,Wallet SM.Estrogen-Induced Monocytic Response Correlates with Temporomandibular Disorder Pain[J].J Dent Res,2017,96(3):285-291[13]Lin X,Kong J,Wu Q,et al.Effect of TLR4/MyD88 signaling pathway on expression of IL-1beta and TNF-alpha in synovial fibroblasts from temporomandibular joint exposed to lipopolysaccharide[J].Mediators Inflamm,2015,2015:329405-[14]Clevers H,Nusse R.Wnt/β-Catenin Signaling and Disease[J].Cell,2012,149(6):1192-1205[15]Bodine PV,Komm BS.Wnt signaling and osteoblastogenesis[J].Rev EndocrMetabDisord,2006,7(1-2):33-39[16]何锦泉,朴正国,欧阳可雄,等.透明质酸对大鼠颞下颌关节滑膜细胞wnt/β-catenin信号通路的影响[J].中国医药指南,2013,(32):299-301[17]杨婷.骨髓间充质干细胞Wnt5a/Ror2信号调控颞下颌关节骨关节病模型大鼠软骨下骨的改建[D]. 第四军医大学,2014[18]Lo WW,Wunder JS,Dickson BC,et al.Involvement and targeted intervention of dysregulated Hedgehog signaling in osteosarcoma[J].Cancer,2014,120(4):537-547[19]Yoshida M,Hata K,Takashima R,et al.The transcription factor Foxc1 is necessary for Ihh-Gli2-regulated endochondral ossification[J].Nat Commun,2015,6:6653-[20]Zhou J,Chen Q,Lanske B,et al.Disrupting the Indian hedgehog signaling pathway in vivo attenuates surgically induced osteoarthritis progression in Col2a1-CreERT2; Ihhfl/fl mice[J].Arthritis Res Ther,2014,16(1):R11-[21]Ishizuka Y,Shibukawa Y,Nagayama M,et al.TMJ degeneration in SAMP8 mice is accompanied by deranged Ihh signaling[J].J Dent Res,2014,93(3):281-287[22]Kanwar J R,Samarasinghe R M,Kumar K,et al.Cissus quadrangularis inhibits IL-1beta induced inflammatory responses on chondrocytes and alleviates bone deterioration in osteotomized rats via p38 MAPK signaling[J].Drug Des DevelTher,2015,9:2927-2940[23]Lim H,Kim H P.Matrix metalloproteinase-13 expression in IL-1beta-treated chondrocytes by activation of the p38 MAPK/c-Fos/AP-1 and JAK/STAT pathways[J].Arch Pharm Res,2011,34(1):109-117[24]马川,陈虹瑜,曲竹丽,等.睡眠剥夺对大鼠颞下颌关节髁突组织p38信号通路相关蛋白表达的影响[J].山东医药,2014,54(6):21-23[25]Ma C,Wu G,Wang Z,et al.Effects of chronic sleep deprivation on the extracellular signal-regulated kinase pathway in the temporomandibular joint of rats[J].PLoS One,2014,9(9):e107544-[26]詹静,吴慧玲,冯剑颖,等.BMP/Smads信号传导通路在兔下颌持续前导后髁突的表达[J].浙江大学学报医学版,2006,35(5):485-490[27]Bechtold TE,Saunders C,Mundy C,et al.Excess BMP Signaling in Heterotopic Cartilage Forming in Prg4-null TMJ Discs[J].J Dent Res,2016,95(3):292-301[28]Lingaraj K,Poh CK,Wang W.Vascular endothelial growth factor (VEGF) is expressed during articular cartilage growth and re-expressed in osteoarthritis[J].Ann Acad Med Singapore,2010,39(5):399-403[29]Pufe T,Harde V,Petersen W,et al.Vascular endothelial growth factor (VEGF) induces matrix metalloproteinase expression in immortalized chondrocytes[J].J Pathol,2004,202(3):367-374[30]Franses RE,McWilliams DF,Mapp PI,et al.Osteochondral angiogenesis and increased protease inhibitor expression in OA[J].Osteoarthritis Cartilage,2010,18(4):563-571[31]Okubo M,Kimura T,Fujita Y,et al.Semaphorin 3A is expressed in human osteoarthritic cartilage and antagonizes vascular endothelial growth factor 165-promoted chondrocyte migration: an implication for chondrocyte cloning[J].Arthritis Rheum,2011,63(10):3000-3009[32]Dong Y,Wu G,Zhu T,et al.VEGF promotes cartilage angiogenesis by phospho-ERK1/2 activation of Dll4 signaling in temporomandibular joint osteoarthritis caused by chronic sleep disturbance in Wistar rats[J].Oncotarget,2017,8(11):17849-17861[33]Yokota T,Shimokawa H,Shibata S,et al.Insulin-like growth factor I regulates apoptosis in condylar cartilage[J].J Dent Res,2008,87(2):159-163[34]江莉婷,魏立,谢银银,等.IGF-1信号通路相关蛋白在大鼠髁突软骨细胞增殖分化中的表达[J].口腔颌面外科杂志,2013,23(3):171-177[35]Zhu Y,Wu G,Zhu G,et al.Chronic sleep restriction induces changes in the mandibular condylar cartilage of rats: roles of Akt,Bad and Caspase-3[J].Int J Clin Exp Med,2014,7(9):2585-2592[36]Chen H,Wu G,Sun Q,et al.Hyperbaric oxygen protects mandibular condylar chondrocytes from interleukin-1beta-induced apoptosis via the PI3K/AKT signaling pathway[J].Am J Transl Res,2016,8(11):5108-5117 |
