| [1]. Sakoda, K., Y. Nakajima, and K. Noguchi, Enamel matrix derivative induces production of vascular endothelial cell growth factor in human gingival fibroblasts. Eur J Oral Sci, 2012. 120(6): p. 513-9.[2]. Villa, O., et al., Subfractions of enamel matrix derivative differentially influence cytokine secretion from human oral fibroblasts. J Tissue Eng, 2015. 6: p. 2041731415575857.[3]. 邹慧儒，秦宗长，张兰成, 釉基质蛋白各组成成分及其作用. 国际腔医学杂志, 2013. 40(3): p. 355-25.[4]. Ono, M., et al., Quest for anti-inflammatory substances using IL-1beta-stimulated gingival fibroblasts. In Vivo, 2011. 25(5): p. 763-8.[5]. Sanchez, G.A., et al., Salivary IL-1beta and PGE2 as biomarkers of periodontal status, before and after periodontal treatment. J Clin Periodontol, 2013. 40(12): p. 1112-7.[6]. Sato, S., et al., Enamel matrix derivative exhibits anti-inflammatory properties in monocytes. J Periodontol, 2008. 79(3): p. 535-40.[7]. Khedmat, S., et al., Effects of enamel matrix derivative on the viability, cytokine secretion, and phagocytic activity of human monocytes. J Endod, 2010. 36(6): p. 1000-3.[8]. Gundersen, R.Y., et al., Systemic administration of enamel matrix derivative to lipopolysaccharide-challenged pigs: effects on the inflammatory response. Surg Infect (Larchmt), 2008. 9(2): p. 161-9.[9]. Giannopoulou, C., et al., Enamel matrix derivative and systemic antibiotics as adjuncts to non-surgical periodontal treatment: biologic response. J Periodontol, 2006. 77(4): p. 707-13.[10]. Almqvist, S., et al., Amelogenins modulate cytokine expression in LPS-challenged cultured human macrophages. Cytokine, 2012. 58(2): p. 274-9.[11]. Myhre, A.E., et al., Anti-inflammatory properties of enamel matrix derivative in human blood. J Periodontal Res, 2006. 41(3): p. 208-13.[12]. Karima, M.M. and T.E. Van Dyke, Enamel matrix derivative promotes superoxide production and chemotaxis but reduces matrix metalloproteinase-8 expression by polymorphonuclear leukocytes. J Periodontol, 2012. 83(6): p. 780-6.[13]. Gassmann, G., et al., Influence of enamel matrix derivative on primary CD4+ T-helper lymphocyte migration, CD25 activation, and apoptosis. J Periodontol, 2009. 80(9): p. 1524-33.[14]. Weinberg, E., et al., Differential effects of prostaglandin E(2) and enamel matrix derivative on the proliferation of human gingival and dermal fibroblasts and gingival keratinocytes. J Periodontal Res, 2010. 45(6): p. 731-40.[15]. Zeldich, E., et al., Enamel matrix derivative induces the expression of tissue inhibitor of matrix metalloproteinase-3 in human gingival fibroblasts via extracellular signal-regulated kinase. J Periodontal Res, 2010. 45(2): p. 200-6.[16]. Zeldich, E., et al., Enamel matrix derivative protects human gingival fibroblasts from TNF-induced apoptosis by inhibiting caspase activation. J Cell Physiol, 2007. 213(3): p. 750-8.[17]. Scheller, J., et al., The pro- and anti-inflammatory properties of the cytokine interleukin-6. Biochim Biophys Acta, 2011. 1813(5): p. 878-88.[18]. Nokhbehsaim, M., et al., Anti-inflammatory effects of EMD in the presence of biomechanical loading and interleukin-1beta in vitro. Clin Oral Investig, 2012. 16(1): p. 275-83.[19]. Nokhbehsaim, M., et al., Effects of enamel matrix derivative on periodontal wound healing in an inflammatory environment in vitro. J Clin Periodontol, 2011. 38(5): p. 479-90.[20]. Deschner, J. and M. Nokhbehsaim, Regulatory effects of inflammatory and biomechanical signals on regenerative periodontal healing. Int J Oral Maxillofac Implants, 2013. 28(6): p. e472-7.[21]. Miron, R.J., et al., Effect of Enamel Matrix Derivative (EMD)-Liquid on Osteoblast and Periodontal Ligament Cell Proliferation and Differentiation. J Periodontol, 2015: p. 1-14.[22]. Miron, R.J., et al., Gene array of primary human osteoblasts exposed to enamel matrix derivative in combination with a natural bone mineral. Clin Oral Investig, 2013. 17(2): p. 405-10. |
