| [1] Wang N, Li H, Lü W, et al. Effects of TiO2 nanotubes with different diameters on gene expression and osseointegration of implants in minipigs[J]. Biomaterials, 2011, 32(29): 6900-6911.[2] Eisenbarth E, Linez P, Biehl V, et al. Cell orientation and cytoskeleton organisation on ground titanium surfaces[J]. Biomolecular Engineering, 2002, 19(2): 233-237.[3] Schwartz Z, Carnes Jr D L, Pulliam R, et al. Porcine fetal enamel matrix derivative stimulates proliferation but not differentiation of pre-osteoblastic 2T9 cells, inhibits proliferation and stimulates differentiation of osteoblast-like MG63 cells, and increases proliferation and differentiation of normal human osteoblast NHOst cells[J]. Journal of Periodontology, 2000, 71(8): 1287-1296.[4] Ponsonnet L, Reybier K, Jaffrezic N, et al. Relationship between surface properties (roughness, wettability) of titanium and titanium alloys and cell behaviour[J]. Materials Science and Engineering: C, 2003, 23(4): 551-560.[5] Kokovic V, Jung R, Feloutzis A, et al. Immediate vs. early loading of SLA implants in the posterior mandible: 5-year results of randomized controlled clinical trial[J]. Clinical Oral Implants Research, 2014, 25(2): e114-e119.[6] van Velzen FJ, Ofec R, Schulten EA, et al. 10-year survival rate and the incidence of peri-implant disease of 374 titanium dental implants with a SLA surface: a prospective cohort study in 177 fully and partially edentulous patients[J]. Clinical Oral Implants Research, 2014 Nov 5. doi: 10.1111/clr.12499.[7] Kim H, Choi S H, Ryu J J, et al. The biocompatibility of SLA-treated titanium implants[J]. Biomedical Materials, 2008, 3(2): 025011.[8] Gong D, Grimes C A, Varghese O K, et al. Titanium oxide nanotube arrays prepared by anodic oxidation[J]. Journal of Materials Research, 2001, 16(12): 3331-3334.[9] Oh S, Daraio C, Chen L H, et al. Significantly accelerated osteoblast cell growth on aligned TiO2 nanotubes[J]. Journal of Biomedical Materials Research Part A, 2006, 78(1): 97-103.[10] Yang W, Xi X, Ran Q, et al. Influence of the titania nanotubes dimensions on adsorption of collagen: An experimental and computational study[J]. Materials Science and Engineering: C, 2014, 34: 410-416.[11] Kubota S, Johkura K, Asanuma K, et al. Titanium oxide nanotubes for bone regeneration[J]. Journal of Materials Science: Materials in Medicine, 2004, 15(9): 1031-1035.[12] Zhao L, Mei S, Chu PK, et al. The influence of hierarchical hybrid micro/nano-textured titanium surface with titania nanotubes on osteoblast functions[J]. Biomaterials, 2010, 31(19): 5072-5082.[13] Meng W, Zhou Y, Zhang Y, et al. Osteoblast behavior on hierarchical micro-/nano-structured titanium surface[J]. Journal of Bionic Engineering, 2011, 8(3): 234-241.[14] 朱青, 焦婷. 钛种植体表面处理对骨整合的影响[J]. 口腔材料器械杂志, 2014, 23(1): 47-49.[15] Cao X, Yu W, Qiu J, et al. RGD peptide immobilized on TiO2 nanotubes for increased bone marrow stromal cells adhesion and osteogenic gene expression[J]. Journal of Materials Science: Materials in Medicine, 2012, 23(2): 527-536.[16] Keselowsky B G, Wang L, Schwartz Z, et al. Integrin α5 controls osteoblastic proliferation and differentiation responses to titanium substrates presenting different roughness characteristics in a roughness independent manner[J]. Journal of Biomedical Materials Research Part A, 2007, 80(3): 700-710.[17] Tamura M, Nemoto E, Sato MM, et al. Role of the Wnt signaling pathway in bone and tooth[J]. Frontiers in Bioscience (Elite edition), 2009, 2: 1405-1413.[18] Galli C, Piemontese M, Lumetti S, et al. Actin cytoskeleton controls activation of Wnt/β-catenin signaling in mesenchymal cells on implant surfaces with different topographies[J]. Acta Biomaterialia, 2012, 8(8): 2963-2968.[19] Wang W, Zhao L, Ma Q, et al. The role of the Wnt/β-catenin pathway in the effect of implant topography on MG63 differentiation[J]. Biomaterials, 2012, 33(32): 7993-8002.[20] McDonald PC, Fielding AB, Dedhar S. Integrin-linked kinase–essential roles in physiology and cancer biology[J]. Journal of Cell Science, 2008, 121(19): 3121-3132.[21] Tan TW, Huang YL, Chang JT, et al. CCN3 increases BMP-4 expression and bone mineralization in osteoblasts[J]. Journal of Cellular Physiology, 2012, 227(6): 2531-2541.[22] Wang W, Liu Q, Zhang Y, et al. Involvement of ILK/ERK1/2 and ILK/p38 pathways in mediating the enhanced osteoblast differentiation by micro/nanotopography[J]. Acta Biomaterialia, 2014, 10(8): 3705-3715.[23] Golden LH, Insogna KL. The expanding role of PI3- kinase in bone[J]. Bone, 2004, 34(1): 3-12[24] Guntur AR, Rosen CJ. The skeleton: a multi-functional complex organ. New insights into osteoblasts and their role in bone formation: the central role of PI3Kinase[J]. Journal of Endocrinology, 2011, 211(2): 123-130.[25] Gu YX, Du J, Si MS, et al. The roles of PI3K/Akt signaling pathway in regulating MC3T3-E1 preosteoblast proliferation and differentiation on SLA and SLActive titanium surfaces[J]. Journal of Biomedical Materials Research Part A, 2013, 101(3): 748-754.[26] Schwartz Z, Lohmann CH, Vocke AK, et al. Osteoblast response to titanium surface roughness and 1α, 25-(OH)2D3 is mediated through the mitogen-activated protein kinase (MAPK) pathway[J]. Journal of Biomedical Materials Research, 2001, 56(3): 417-426.[27] Sisk MA, Lohmann CH, Cochran DL, et al. Inhibition of cyclooxygenase by indomethacin modulates osteoblast response to titanium surface roughness in a time-dependent manner[J]. Clinical Oral Implants Research, 2001, 12(1): 52-61.[28] Dean DD, Campbell CM, Gruwell SF, et al. Arachidonic acid and prostaglandin E2 influence human osteoblast (MG63) response to titanium surface roughness[J]. Journal of Oral Implantology, 2008, 34(6): 303-312. |
