[1] |
MarazitaML.The Evolution of Human Genetic Studies of Cleft Lip and Cleft Palate[J]. Annu Rev Genomics Hum Genet, 2012, 13:263-283.
|
[2] |
Leslie EJ, MarazitaML.Genetics of cleft lip and cleft palate[J].Am J Med Genet C Semin Med Genet, 2013, 163C(4):246-258
|
[3] |
Watkins SE, Meyer RE, Strauss RP, Aylsworth AS.Classification,
|
[4] |
epidemiology, andgenetics of orofacial clefts[J].Clinics in plastic surgery,
|
[5] |
14, 41(2):149-163.
|
[6] |
Hozyasz KK, Mostowska A, Wójcicki P, et al.Nucleotide Variants of the BH4 Biosynthesis Pathway Gene GCH1 and the Risk of Orofacial Clefts[J].MolNeurobiol, 2016, 53(1):769-776
|
[7] |
Akaike H.A New Look at the Statistical Model Identification[J]. IEEE-Automatic Control, IEEE Transactions, 1974, AC-19:716-723.
|
[8] |
Rahimov F, Jugessur A, Murray JC.Genetics of nonsyndromic orofacial clefts[J].Cleft Palate Craniofac J, 2012, 49(1):73-91
|
[9] |
Group I W.Prevalence at Birth of Cleft Lip With or Without Cleft Palate[J], Data From the International Perinatal Database of Typical Oral Clefts (IPDTOC), 2011, 48(1):66-81.
|
[10] |
Allagh KP, Shamanna BR, Murthy GV, et al.Birth prevalence of neural tube defects and orofacial clefts in India: a systematic review and meta-
|
[11] |
Analysis[J].PLOS ONE, 2015, 10(3):e0118961.
|
[12] |
Butali A, Adeyemo WL, Mossey PA, et al.Prevalence of orofacial clefts in Nigeria[J].The Cleft palate-craniofacial journal, 2014, 51(3):320-325
|
[13] |
中国出生缺陷防治报告(2012)[R].北京:中华人民共和国卫生部,2012.
|
[14] |
Younkin SG, Scharpf RB, Schwender H, et al.A genome-wide study of de novo deletions identifies a candidate locus for non-syndromic isolated cleft lip/palate risk[J]. BMC Genet, 2014, 15:24.
|
[15] |
Keil KP, Vezina CM.DNA methylation as adynamic regulator of development and disease processes:spotlight on the prostate[J].Epigenomics, 2015, 7(3):413-425
|
[16] |
Wahlqvist ML, KrawetzSA, RizzoNS, et al.Early-life influences on obesity: from preconception toadolescence[J]. Ann N YAcad Sci, 2015, 1347:1-28.
|
[17] |
El Hajj N, SchneiderNE, LehnenH, et al.Epigenetics and life-long consequences of an adversenutritional and diabetic intrauterine environment[J].Reproduction.2014, 148(6):111-120.
|
[18] |
Lupo PJ, Chapa C, NousomeD, et al.National Birth Defects Prevention Study.A GCH1 haplotype and risk of neural tube defects in theNational Birth Defects Prevention Study[J]. Mol Genet Metab, 2012, 107(3):
|
[19] |
2-595.
|
[20] |
West JR, Blake CA.Fetal alcohol syndrome: an assessment of the field[J].Exp Biol Med, 2005, 230(6):354-356
|
[21] |
DeRoo LA, Wilcox AJ, Lie RT, et al.Maternal alcohol binge-drinking in the first trimester and the risk of orofacial clefts in offspring: a large population based pooling study[J].Eur J Epidemiol, 2016, 31(10):1021-
|
[22] |
1034.
|
[23] |
Honein MA, Devine O, Grosse SD, et al.Prevention of orofacial clefts caused by smoking:implications of the Surgeon General’s report[J].Birth Defects Res A Clin Mol Teratol, 2014, 100(11):822-825
|
[24] |
Kummet CM, Moreno LM, Wilcox AJ, et al.Passive Smoke Exposure as a Risk Factor for Oral Clefts-A Large International Population-Based Study[J].Am J Epidemiol, 2016, 183(9):834-841
|
[25] |
Keil KP, VezinaCM.DNA methylation as adynamic regulator of development and disease processes:spotlight on the prostate[J].Epigenomics, 2015, 7(3):413-425
|
[26] |
Wahlqvist ML, KrawetzSA, RizzoNS, et al.Early-life influences on obesity: from preconception toadolescence[J]. Ann N YAcad Sci, 2015, 1347:1-28.
|