[1] Sczepanik FSC, Grossi ML, Casati M,et al. Periodontitis is an inflammatory disease of oxidative stress:We should treat it that way[J]. Periodontol 2000, 2020, 84(1):45-68. [2] Kanzaki H, Wada S, Narimiya T,et al. Pathways that regulate ROS scavenging enzymes, and their role in defense against tissue destruction in periodontitis[J]. Front Physiol, 2017, 8:351. [3] Chevion M, Berenshtein E, Stadtman ER. Human studies related to protein oxidation:Protein carbonyl content as a marker of damage[J]. Free Radic Res, 2000, 33(Suppl):S99-108. [4] Dalle-Donne I, Giustarini D, Colombo R,et al. Protein carbonylation in human diseases[J]. Trends Mol Med, 2003, 9(4):169-176. [5] Bradford MM. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding[J]. Anal Biochem, 1976, 72(1/2):248-254. [6] Liu ZQ, Liu Y, Song YQ,et al. Systemic oxidative stress biomarkers in chronic periodontitis:A meta-analysis[J]. Dis Markers, 2014, 2014:931083. [7] LiuC, Mo L, Niu Y, et al. The role of reactive oxygen species and autophagy in periodontitis and their potential linkage[J]. Front Physiol, 2017, 8:439. [8] Canakçi CF, Tatar A, Canakçi V,et al. New evidence of premature oxidative DNA damage:Mitochondrial DNA deletion in gingival tissue of patients with periodontitis[J]. J Periodontol, 2006, 77(11):1894-1900. [9] Enache TA, Matei E, Diculescu VC. Electrochemical sensorfor carbonyl groups in oxidized proteins[J]. Anal Chem, 2019, 91(3):1920-1927. [10] Akagawa M. Protein carbonylation:Molecular mechanisms, biological implications, and analytical approaches[J]. Free Radic Res, 2021, 55(4):307-320. [11] Sharma A, Weber D, Raupbach J,et al. Advanced glycation end products and protein carbonyl levels in plasma reveal sex-specific differences in Parkinson's and Alzheimer's disease[J]. Redox Biol, 2020, 34:101546. [12] Mateen S, Moin S, Khan AQ,et al. Increased reactive oxygen species formation and oxidative stress in rheumatoid arthritis[J]. PLoS One, 2016, 11(4):e0152925. [13] Hecker M, Wagner AH. Role of protein carbonylation in diabetes[J]. J Inherit Metab Dis, 2018, 41(1):29-38. [14] Gryszczyńska B, Formanowicz D, Budzyń M,et al. Advanced oxidation protein products and carbonylated proteins as biomarkers of oxidative stress in selected atherosclerosis-mediated diseases[J]. Biomed Res Int, 2017, 2017:4975264. [15] Khorasani S, Boroumand N,Ghaeni Pasavei A, et al. A study on association between protein carbonyl and anti-cyclic citrullinated peptide antibody in rheumatoid arthritis:Introducing a new supplementary biomarker[J]. Indian J Clin Biochem, 2020, 35(3):347-352. [16] 陈崇崇, 钟良军. 龈沟液生物标志物在慢性牙周炎诊疗中的研究进展[J]. 口腔医学, 2019, 39(11):1047-1052. [17] Baltacioğlu E, Akalin FA, Alver A,et al. Protein carbonyl levels in serum and gingival crevicular fluid in patients with chronic periodontitis[J]. Arch Oral Biol, 2008, 53(8):716-722. [18] Griffiths GS. Formation, collection and significance ofgingival crevice fluid[J]. Periodontol 2000, 2003, 31:32-42. [19] Wei D, Zhang XL, Wang YZ,et al. Lipid peroxidation levels, total oxidant status and superoxide dismutase in serum, saliva and gingival crevicular fluid in chronic periodontitis patients before and after periodontal therapy[J]. Aust Dent J, 2010, 55(1):70-78. [20] Sculley DV, Langley-Evans SC. Periodontal disease is associated with lower antioxidant capacity in whole saliva and evidence of increased protein oxidation[J].Clin Sci (Lond), 2003, 105(2):167-172. [21] Pradeep AR, Ramchandraprasad MV, Bajaj P,et al. Protein carbonyl:An oxidative stress marker in gingival crevicular fluid in healthy, gingivitis, and chronic periodontitis subjects[J]. Contemp Clin Dent, 2013, 4(1):27-31. [22] Lutfioğlu M, Aydoğdu A, Atabay VE,et al. Gingival crevicular fluid oxidative stress level in patients with periodontal disease and hyperlipidemia[J]. Braz Oral Res, 2017, 31:e110. [23] Nguyen TT, Ngo LQ, Promsudthi A,et al. Salivary oxidative stress biomarkers in chronic periodontitis and acute coronary syndrome[J]. Clin Oral Investig, 2017, 21(7):2345-2353. [24] Rokad F, Moseley R, Hardy RS,et al. Cerebral oxidative stress and microvasculature defects in TNF-α expressing transgenic and Porphyromonas gingivalis-infected ApoE-/- mice[J]. J Alzheimer's Dis, 2017, 60(2):359-369. [25] Chapple IL, Brock GR, Milward MR,et al. Compromised GCF total antioxidant capacity in periodontitis:Cause or effect?[J]. J Clin Periodontol, 2007, 34(2):103-110. |