[1] Kinane DF, Stathopoulou PG, Papapanou PN. Periodontal diseases[J]. Nat Rev Dis Primers, 2017, 3: 17038. [2] Hajishengallis G, Chavakis T. Local and systemic mechanisms linking periodontal disease and inflammatory comorbidities[J]. Nat Rev Immunol, 2021, 21(7):426-440. [3] 闫福华. 牙周炎对全身疾病和健康影响的研究进展[J]. 口腔医学, 2018, 38(7):577-581. [4] Sun K,宋忠臣. 慢性牙周炎与慢性肾病相关性研究进展[J]. 口腔医学, 2020, 40(1):59-62. [5] Kose O, Kurt Bayrakdar S, Unver B, et al. Melatonin improves periodontitis-induced kidney damage by decreasing inflammatory stress and apoptosis in rats[J]. J Periodontol, 2021, 92(6):22-34. [6] França LFC, Vasconcelos ACCG, da Silva FRP, et al. Periodontitis changes renal structures by oxidative stress and lipid peroxidation[J]. J Clin Periodontol, 2017, 44(6):568-576. [7] Sharma P, Fenton A, Dias IHK, et al. Oxidative stress links periodontal inflammation and renal function[J]. J Clin Periodontol, 2021, 48(3):357-367. [8] Lin TN, Wu VCC, Wang CY. Autophagy in chronic kidney diseases[J]. Cells, 2019, 8(1):61. [9] 莫龙义, 贾小玥, 刘程程, 等. 细胞自噬在牙周炎中的作用与机制[J]. 华西口腔医学杂志, 2019, 37(4):422-427. [10] Kimura T, Isaka Y, Yoshimori T. Autophagy and kidney inflammation[J]. Autophagy, 2017, 13(6):997-1003. [11] Greabu M, Giampieri F, Imre MM, et al. Autophagy, one of the main steps in periodontitis pathogenesis and evolution[J]. Molecules, 2020, 25(18):4338. [12] Liu CC, Mo LY, Niu YL, et al. The role of reactive oxygen species and autophagy in periodontitis and their potential linkage[J]. Front Physiol, 2017, 8: 439. [13] Kaushal GP, Chandrashekar K, Juncos LA. Molecular interactions between reactive oxygen species and autophagy in kidney disease[J]. Int J Mol Sci, 2019, 20(15):3791. [14] Choi ME. Autophagy in kidney disease[J]. Annu Rev Physiol, 2020, 82: 297-322. [15] Fantus D, Rogers NM, Grahammer F, et al. Roles of mTOR complexes in the kidney: Implications for renal disease and transplantation[J]. Nat Rev Nephrol, 2016, 12(10):587-609. [16] 岳轶云, 丁小函, 李艳, 等. Toll样受体4在慢性牙周炎诱发大鼠肝脏炎症中的作用[J]. 口腔医学研究, 2020, 36(2):139-142. [17] 李艳, 夏博园, 李鑫, 等. Nrf2在牙周炎诱发大鼠肝损伤中的作用研究[J]. 口腔医学研究, 2020, 36(12):1113-1116. [18] Kitamura M, Mochizuki Y, Miyata Y, et al. Pathological characteristics of periodontal disease in patients with chronic kidney disease and kidney transplantation[J]. Int J Mol Sci, 2019, 20(14):3413. [19] Chopra A, Sivaraman K. An update on possible pathogenic mechanisms of periodontal pathogens on renal dysfunction[J]. Crit Rev Microbiol, 2019, 45(5/6):514-538. [20] Chambrone L, Foz AM, Guglielmetti MR, et al. Periodontitis and chronic kidney disease: A systematic review of the association of diseases and the effect of periodontal treatment on estimated glomerular filtration rate[J]. J Clin Periodontol, 2013, 40(5):443-456. [21] Horikoshi S, Fukuda N, Tsunemi A, et al. Contribution of TGF-β1 and effects of gene silencer pyrrole-imidazole polyamides targeting TGF-β1 in diabetic nephropathy[J]. Molecules, 2020, 25(4):950. [22] Galeno JG, França L, da Silva FRP, et al. Renal alterations caused by ligature-induced periodontitis persist after ligature removal in rats[J]. J Periodontal Res, 2021, 56(2):306-313. [23] Tang CY, Livingston MJ, Liu ZW, et al. Autophagy in kidney homeostasis and disease[J]. Nat Rev Nephrol, 2020, 16(9):489-508. [24] Liu H, Wang Q, Shi G, et al. Emodinameliorates renal damage and podocyteinjury in a rat model of diabetic nephropathy via regulating AMPK/mTOR-mediated autophagy signaling pathway[J]. Diabetes Metab Syndr Obes, 2021, 14: 1253-1266. |